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Ferroptosis in myocardial I/R injury Ferroptosis is the predominant form of cell death in the prolonged reperfusion phase of myocardial I/R Injury [22]

Using primary hepatocytes and peritoneal macrophages, the authors demonstrated that a mixture of 17,18-EEQ, 5-HEPE, and 9-HEPE attenuated the palmitate-induced activation of pro-inflammatory cytokines and the Jun N-terminal kinase (JNK) pathway

Natural killer (NK) cell cytotoxicity increased significantly in the high-dose group compared to the placebo group at three months, indicating a potential enhancement of immune function with GSH supplementation

Beneficial therapeutic effects of Nigella sativa and/or Zingiber officinale in HCV patients in Egypt